By Sinatra R.S., Ginsberg B., Viscusi E.
This textbook presents an outline of discomfort administration worthy to experts in addition to non-specialists, surgeons, and nursing employees.
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97. Ding JD, Weinberg RJ. Localization of soluble guanylyl cyclase in the superficial dorsal horn. Journal Comp Neurol. 2006;495(6):668–678. 98. Ikeda H, Kusudo K, Murase K. Nitric oxide-dependent long-term potentiation revealed by real-time imaging of nitric oxide production and neuronal excitation in the dorsal horn of rat spinal cord slices. Eur J Neurosci. 2006;23(7):1939–1943. 99. Wang Z, Gardell LR, Ossipov MH, et al. Pronociceptive actions of dynorphin maintain chronic neuropathic pain. J Neurosci.
Waldhoer M, Bartlett SE, Whistler JL. Opioid receptors. Ann Rev Biochem. 2004;73:953–990. Pain Pathways and Acute Pain Processing 57. Raynor K, Kong H, Chen Y, et al. Pharmacological characterization of the cloned kappa-, delta-, and mu-opioid receptors. Mol Pharmacol. 1994;45(2):330–334. 58. Margas W, Zubkoff I, Schuler HG, Janicki PK, Ruiz-Velasco V. Modulation of Ca2+ channels by heterologously expressed wildtype and mutant human micro-opioid receptors (hMORs) containing the A118G single-nucleotide polymorphism.
96 Ding and Weinberg97 found that NK1 receptors in lamina I were the major target for NO. 96 Superoxide has also been identified as a novel mediator of central sensitization. 99 In the inflammatory process, superoxide is produced at such a rapid rate that SOD is unable to remove it. 100 Wang and coworkers100 found that the synthetic compound (M40403) that mimics SOD prevents the development of inflammation and hyperalgesia after injection of noxious mediators. 100 Furthermore, Muscoli et al101 showed that SO may contribute to various forms of pain events that are driven by NMDA receptor activation.